A recent email from Mark Purdey to the farmtalking forum(wth Mark Purdey's permission and edits) August 30 2004
It is often the 'grass roots' people who have made the creative leaps in scientific thinking and discoveries.
This is all too often ignored for many decades after the initial flashburst of insight - until some socially recognised 'expert' stumbles on the same idea. Meanwhile society has been disadvantaged for all of those decades in that it has not derived any of the social benefits from that discovery - owing to the arrogant / bigoted elements of the scientific leadership that automatically reject any ideas flowing in from the non scientific quarters of the community.
So communication has to be the key - a crossflow of knowledge passing between academic and practical / hands-on communities in the world. That sows the seeds for the growth of lateral thinking which will inevitably result in positive scientific advancements.
But you are not going to communicate to 99% of the population if you confine your communications to the elitist phraseology of the scientific world. Some so called 'experts' hide their inadequacies behind that veil of scientific jargon, and they end up suffocating themselves in meaningless phraseology; which, once you have decoded it, turns out to be saying absolutely nothing constructive at all - often wholly unscientific too.
I always remember the spate of articles appeared in the Veterinary Record and other prestiguous journals about the outbreak of BSE in Kudu antelope . One of the explanations was generated by Ray Bradley ( the former government's chief BSE coordinator ) Because, no meat and bone meal had been fed to these antelope in any guise at all - only dried grass / alfalfa pellets and minerals - and this was embarrassing to the official theory, the authors had launched into a ridiculously far fetched ramble of epidemiological jargon which was trying to suggest that in the history of the zoo, some cow incubating BSE might have actually walked across the paddock where the kudu were kept and deposited prions on the pasture in their dung.
I could not believe such stupidity.
Have they ever kept the domestic cow at the London Zoo? I would not pay the entry fee to the zoo to go and see a common Friesian. Also, how many runaway stray cows do you get in Regent's Park in the centre of London ?
Then an equally ridiculous hypothesis - how wild cats had access to the meat store which was the source of the Pumas' / Cheetahs' feed ( they had developed BSE too ), so those cats might have cross contaminated the Kudu feed store with the infectious prion agent - where the wild cats also visited. It went on trying to squeeze blood out of a sinking stone......
If I had even submitted ( let alone got published ) such an unscientific hypothesis to the Vet Record, I would have been discredited for life - debarred from getting published or even considered for publication ever again. But because the unscientific myth of these authors was supportive of the government theory, this "landmark" publication is still upheld as the unquestionable gospel according to the cause of BSE in the zoo animals , even today. This kind of junk science has thwarted the whole healthy evolution of scientific advancement in the world , and it must be brought to account immediately.
It is clear that BSE is not going to present us with the crisis of biblical proportions that the leading experts had forecast. But one day the ignorant, unilateral, self protecting approach that is currently adopted by Western governments into dealing with mystery epidemics might actually backfire on us - particularly if a real, all life threatening condition does actually rear its ugly head one day. This could lead to the extermination of life on earth. So we need to change the way that governments approach and deal with these kind of crises NOW, before it is too late.
from an earlier email...The issue of copper depletion / disruption as a TSE causal prerequisite which was first raised in the academic literature in my paper that reported the data from my 1995 - 1998 analytical studies of the food chains where sporadic TSEs were clustering around the world.
In this paper I combined the knowledge already brought to us by Hornshaw / Brown et al with that of my own environmental data, to hypothesise that the displacement or loss of copper from the prion protein molecule in conjunction with the substitution by a rogue replacement metal - e.g.; manganese or silver, etc - caused the malformed protease resistant form of prion protein to develop ( e.g. the abnormal form that is seen in the brains of TSE victims).
Whilst someone had proposed copper toxicity as a cause for TSE, nobody had proposed copper loss as a prerequisite before, which I found strange since the chemical cuprizone had been repeatedly used by the Compton labs during the 1970s to induce a chemical scrapie in mice ( albeit non-transmissible, since, according to my theory, there was no additional rogue metal microcrystal prerequisite involved in these tests that would have rendered the scrapie transmissible;). I guess that the researchers did not see the possible aetiological relevance of the fact that cuprizone is a copper chelator. This naturally interested me when I started to observe rock bottom levels copper in all of the TSE cluster foodchains I was visiting and sampling around the world ( e.g.; where the TSE affected populations were self sufficient off their local foodchain ), and led to me generating this theory of copper loss from the prion protein.
Brown et al, then tested my hypothesis of high manganese / low copper in cell cultures and got a positive result. He produced the protease resistant prion using my hypothesis - and this elevated my theory several rungs up the ladder of respectability amongst the non political quarters of the purist scientific community. For no research team had ever produced protease resistant prion protein before - as a de novo transformation. This deserved a lot more academic attention than it actually received..."
Having looked at 46 different metals, I have pinpointed a clear cut correlation with the excessive levels of two toxic metals in the soils / vegetation ( and their radioactive emissions) that I collected from the farm where the BSE cow was raised and the CWD affected deer farms, etc, in Alberta / Saskatchewan and the other TSE hotspot zones that I sampled across Colorado / Wisconsin / New Mexico..."
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